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In addition to the mechanisms proposed above by which induction of Nrf2-target genes might accelerate the promotion and progression of cancer, a convincing case could be argued that upregulation of Nrf2 during tumorigenesis provides cancer cells with additional diversity, possibly by allowing them to be reprogrammed toward pluripotency, which might increase their ability to evolve further

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10.1016/s0167-4889(00)00119-1 [DOI] [PubMed] [Google Scholar] Rahman, M