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Impaired Hemostasis and the Role of Oxidative Stress-Induced Signaling in Thrombotic Disorders—Where We Stand Today

No human clinical trials have been published for either compound

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One further important difference between second messenger-dependent protein kinase- and GRK-mediated phosphorylation of GPCRs is that activated PKA and PKC are in some instances able to phosphorylate agonist-unoccupied GPCRs, indicating that this type of phosphorylation could underlie some forms of heterologous desensitization (Clark et al ., 1988)